Arc/Arg3.1 Is Essential for the Consolidation of Synaptic Plasticity and Memories

نویسندگان

  • Niels Plath
  • Ora Ohana
  • Björn Dammermann
  • Mick L. Errington
  • Dietmar Schmitz
  • Christina Gross
  • Xiaosong Mao
  • Arne Engelsberg
  • Claudia Mahlke
  • Hans Welzl
  • Ursula Kobalz
  • Anastasia Stawrakakis
  • Esperanza Fernandez
  • Robert Waltereit
  • Anika Bick-Sander
  • Eric Therstappen
  • Sam F. Cooke
  • Veronique Blanquet
  • Wolfgang Wurst
  • Benedikt Salmen
  • Michael R. Bösl
  • Hans-Peter Lipp
  • Seth G.N. Grant
  • Tim V.P. Bliss
  • David P. Wolfer
  • Dietmar Kuhl
چکیده

Arc/Arg3.1 is robustly induced by plasticity-producing stimulation and specifically targeted to stimulated synaptic areas. To investigate the role of Arc/Arg3.1 in synaptic plasticity and learning and memory, we generated Arc/Arg3.1 knockout mice. These animals fail to form long-lasting memories for implicit and explicit learning tasks, despite intact short-term memory. Moreover, they exhibit a biphasic alteration of hippocampal long-term potentiation in the dentate gyrus and area CA1 with an enhanced early and absent late phase. In addition, long-term depression is significantly impaired. Together, these results demonstrate a critical role for Arc/Arg3.1 in the consolidation of enduring synaptic plasticity and memory storage.

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عنوان ژورنال:
  • Neuron

دوره 52  شماره 

صفحات  -

تاریخ انتشار 2006